BI 103
Lecture Notes: Chapter 32 Immune System: Defense against Disease
I. Barriers
(External Defense)
A) Skin
1. dry, nutrient free
zone 2. natural
antibiotics 3. skin
continually replaced
B) Mucous Membranes (Line digestive,
respiratory, urogenital tracts: produce mucus)
1. Secrete mucus to trap
enzymes, contains antibacterial enzymes
2. Cilia sweep up mucus
II. Non-
specific Internal Defenses
A)
Phagocytic Cells: Engulf bacteria, damaged
cells by phagocytosis
1. Macrophages 2. Neutrophils
B) Natural Killer Cells: disrupt
cell membranes of invaders
C) Inflammation: Damaged cells
1. Signal mast cells to
release histamine
2. Initiate blood
clotting
3. Attract macrophages
and neutrophils
4. Signal pain to CNS
D) Fever
1. Increases macrophage activity
and antibody production
2. Slows bacterial
reproduction
III.
Specific internal defenses: Immune Response
A) Key Players: specialized
leukocytes = lymphocytes
(see table 32-1)
1. B Cells 2. T Cells
B) Fundamental Steps in the Immune
Response
1. Immune system must
recognize invader
A. Antigens:
molecules located on surface of cells which triggers an immune
response (on bacteria, on infected body cells, full macrophages)
1.
B cells produce antibodies which recognize antigens
a.
“Y” shaped proteins, high specificity binding site
2.
T cells produce T-cell receptors (stay bound to T cell)
B. Body
recognizes “self” cells via major histocompatibility
complex
1.
unique sets of proteins/polysaccharides on cell surface
2. Immune system must
launch attack
A. Humoral immunity (B cells and circulating antibodies)
**attack
microbes before they have invaded body cells
1.
B cell antibody receptor binds antigen
2.
Now activated B cell divides rapidly (clonal
selection)
a)
produces memory cells
b)
produces plasma cells (antibody factories)
3. Antibodies destroy invaders (and attract phagocytic cells)
a)
inactivate invader by binding all over it
b)
clump invaders together
c)
coat invaders with blood proteins
d) all
of these increase phagocytic cell activity
B. Cell
mediated immunity
(Three types
of T cells, produced after receptor on T cell binds to antigen)
**Attack
microbes after they have invaded body cells
1. Cytotoxic
T cells: work like natural killer cells
2.
Helper T cells: stimulate immune cell division
3.
Memory T cells: persist past infection
3. Immune system must
remember past victories
A. Memory
cells “remember” specific antigens
B. Respond
rapidly to repeat invasion
IV. Medical
Care Augments Immune Response
A)
Antibiotics: Slow down microbial
(NOT VIRUSES) reproduction
B)
Vaccinations: Inject weakened/killed microbes: stimulate memory cell
production
V.
Malfunctions of the immune system
A) Allergies: B cells recognize
harmless substances as antigen
B) Autoimmune diseases
1. Type I Diabetes
2. Multiple Sclerosis